研究者業績

田原 麗衣

タハラ レイ  (Rei Tahara)

基本情報

所属
成蹊大学 経営学部 総合経営学科 准教授
学位
修士(体育学)(2014年3月 筑波大学)

研究者番号
50794286
J-GLOBAL ID
202001015170579520
researchmap会員ID
R000001789

委員歴

 1

論文

 6
  • 柴田 聡, 田原 麗衣, 谷口 裕美子, 杉沼 春美, 渡部 厚一, 藤井 範久
    トレーニング科学 36(1) 81-92 2024年4月  査読有り
  • Ryota Sone, Keisei Kosaki, Rei Tahara, Koichi Watanabe, Koichiro Hayashi
    Asian Journal of Sports Medicine 14(4) 2023年10月14日  査読有り
    Background: Excessive production of salivary nitric oxide (s-NO) is a risk factor for oral diseases such as Sjögren syndrome and periodontal disease. Although s-NO is known to be affected by exercise, it is unclear how the difference in exercise intensities affects the s-NO response. Objectives: The purpose of this study was to investigate the acute effect of different exercise intensities (high vs. low intensity) on s-NO production using a cross-over design. Methods: Five healthy male young adults (21.6 ± 0.9 years) performed two ergometer exercises for 30 min at different exercise intensities: 50% heart rate (HR)reserve (low-intensity condition) and 80% HRreserve (high-intensity condition). Saliva samples were collected before (pre) and after (post 0-h and post 1-h) exercises. Results: The s-NO concentration significantly increased (P < 0.05) after the high-intensity exercise (∆changes in post 0-h; +244 ± 53 µmol/L, post 1-h; +352 ± 58 µmol/L), but the s-NO concentration did not change after the low-intensity exercise (∆changes in post 0-h; +11 ± 37 µmol/L, post 1-h; +64 ± 69 µmol/L). Conclusions: This study reveals that s-NO production is dependent on exercise intensities, suggesting that low-intensity exercise can be possible without increasing the risk of oral diseases.
  • 田原麗衣, 上田泰
    成蹊大学一般研究報告 53(4) 2023年3月  筆頭著者
  • 柴田聡, 田原麗衣, 萩原正大, 渡部厚一, 藤井範久
    11 12-24 2023年1月  査読有り
  • Ryota Sone, Kai Matsuba, Rei Tahara, Nobuhiko Eda, Keisei Kosaki, Subrina Jesmin, Shumpei Miyakawa, Koichi Watanabe
    Journal of clinical medicine research 11(2) 114-120 2019年2月  査読有り
    Background: High-intensity exercise affects the level of salivary nitric oxide (NO) with an impact on oxidative stress such as a reactive nitrogen-oxide species. However, in athletes with high-intensity training, the relationship between salivary NO levels and oxidative stress is yet to be clear. Additionally, the association of salivary NO levels and the common health disorders of athletes is unknown. Thus, the aim of this cross-sectional study was to clarify the relationship between salivary NO levels and oxidative stress, and the health/medical disorders existing in elite class university athletes. Methods: In 250 athletes (males, 151 and females, 99) from undergraduate levels of Japanese University, we investigated the relationship between levels of salivary NO and oxidative stress markers: derived reactive oxygen species (d-ROMs) and biological antioxidant potential (BAP), and also examined that whether salivary NO levels are associated with diseases. Results: There were no significant association between the levels of salivary NO and oxidative stress markers (such as d-ROM and BAP). From the questionnaire, asthma was the most prevalent as evident from medical history of the athletes. Additionally, the salivary NO levels were higher (520 ± 43 µmol/L vs. 375 ± 13 µmol/L, P < 0.05) in the asthma group (n = 9) than in the non-asthma group (n = 241). We determined the optimal cut-off value (P = 0.019) of the salivary NO levels for asthma was 425 µmol/L, with a sensitivity of 88.9% and specificity of 61.8% (area under the curve (AUC), 0.73). Conclusions: Our results suggest that the high levels of salivary NO in trained university athletes in Japan may potentially predict asthma. And this salivary NO level is not associated with markers of oxidative stress and existing diseases in athletes studied here.
  • Kazuhito Watanabe, Masashi Ichinose, Rei Tahara, Takeshi Nishiyasu
    AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY 306(2) H251-H260 2014年1月  査読有り
    We tested the hypotheses that, in humans, changes in cardiac output (CO) and total peripheral vascular resistance (TPR) occurring in response to isometric handgrip exercise vary considerably among individuals and that those individual differences are related to differences in muscle metaboreflex and arterial baroreflex function. Thirty-nine healthy subjects performed a 1-min isometric handgrip exercise at 50% of maximal voluntary contraction. This was followed by a 4-min postexercise muscle ischemia (PEMI) period to selectively maintain activation of the muscle metaboreflex. All subjects showed increases in arterial pressure during exercise. Interindividual coefficients of variation (CVs) for the changes in CO and TPR between rest and exercise periods (CO: 95.1% and TPR: 87.8%) were more than twofold greater than CVs for changes in mean arterial pressure (39.7%). There was a negative correlation between CO and TPR responses during exercise (r = -0.751, P &lt; 0.01), but these CO and TPR responses correlated positively with the corresponding responses during PEMI (r = 0.568 and 0.512, respectively, P &lt; 0.01). The CO response during exercise did not correlate with PEMI-induced changes in an index of cardiac parasympathetic tone and cardiac baroreflex sensitivity. These findings demonstrate that the changes in CO and TPR that occur in response to isometric handgrip exercise vary considerably among individuals and that the two responses have an inverse relationship. They also suggest that individual differences in components of the pressor response are attributable in part to variations in muscle metaboreflex-mediated cardioaccelerator and vasoconstrictor responses.

講演・口頭発表等

 6

共同研究・競争的資金等の研究課題

 1